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Archive for March, 2011

ALLERGY-ARTHRITIS CONNECTION: CLINICAL ECOLOGISTS DETERMINING CAUSES

Some cases of arthritis and some cases of non-arthritic ecologic diseases (affecting other parts and systems of the body) are so complicated that they positively require the expertise of a well-trained, ecologically oriented physician. A regular allergist – even a board-certified specialist – does not have the knowledge and skill to help you.
Traditionally trained allergists have never been exposed to, or have not taken the opportunity to become familiar with, the currently available information concerning the often easy to demonstrate causes of environmentally produced arthritis. Few medical schools teach anything at all about ecologic science (the clinical application of ecologic principles), and there is not very much instruction about allergy, preventive medicine, or clinical nutrition given to our future doctors.
It is understandable how many fine, sincere physicians are not nearly as effective as they could be despite their intellectual capacity and desire to help their patients. Their diagnostic and therapeutic horizons are greatly limited by these generally unrecognized deficiencies in their training.
The Arthritis Foundation, which is a major source of information for interested physicians and the public, has flatly rejected the idea that arthritis could be an allergic disorder of the joints and muscles. The foundation does not or will not believe that arthritis might be associated with, or caused by reactions to or a lack of, certain substances in the diet. The foundation does believe that an allergy-like immunologic reaction caused by factors unknown to them may be responsible for arthritis, but they continue to ignore the dietary and environmental factors that ecologists have clearly shown in thousands of instances are capable of setting off attacks of arthritis. This is a very puzzling inconsistency.
Be that as it may, you can judge the facts for yourself and make up your own mind.
Every disease must have a cause. A good way of determining that our testing for the causes of arthritis by provocative techniques is accurate and clinically effective is to see if application of this information leads to a treatment that cures or significantly relieves an active case or prevents its recurrence. That is exactly what clinical ecologists have done for many years in case after case and study after study with thousands of patients. We do not claim to be able to cure the bone damage associated with arthritis if the allergy-initiated inflammation and structural changes are allowed to continue too long, but we can give considerable relief to many. Together, the arthritic and the ecologist often cure arthritis by continuous attention to the demonstrable causes in each case, while the condition remains in its reversible state. Careful avoidance of test-identified food offenders in conjunction with a rotary-diversified diet, desensitization treatment (immunotherapy), and changes in life-style, along with nutritional supplements, can prevent the occurrence of future “spontaneous” flare-ups due to exposure to previously unsuspected dietary and environmental factors.
*15/295/5*

ALLERGY-ARTHRITIS CONNECTION: CLINICAL ECOLOGISTS DETERMINING CAUSES Some cases of arthritis and some cases of non-arthritic ecologic diseases (affecting other parts and systems of the body) are so complicated that they positively require the expertise of a well-trained, ecologically oriented physician. A regular allergist – even a board-certified specialist – does not have the knowledge and skill to help you.Traditionally trained allergists have never been exposed to, or have not taken the opportunity to become familiar with, the currently available information concerning the often easy to demonstrate causes of environmentally produced arthritis. Few medical schools teach anything at all about ecologic science (the clinical application of ecologic principles), and there is not very much instruction about allergy, preventive medicine, or clinical nutrition given to our future doctors.It is understandable how many fine, sincere physicians are not nearly as effective as they could be despite their intellectual capacity and desire to help their patients. Their diagnostic and therapeutic horizons are greatly limited by these generally unrecognized deficiencies in their training.The Arthritis Foundation, which is a major source of information for interested physicians and the public, has flatly rejected the idea that arthritis could be an allergic disorder of the joints and muscles. The foundation does not or will not believe that arthritis might be associated with, or caused by reactions to or a lack of, certain substances in the diet. The foundation does believe that an allergy-like immunologic reaction caused by factors unknown to them may be responsible for arthritis, but they continue to ignore the dietary and environmental factors that ecologists have clearly shown in thousands of instances are capable of setting off attacks of arthritis. This is a very puzzling inconsistency.Be that as it may, you can judge the facts for yourself and make up your own mind.Every disease must have a cause. A good way of determining that our testing for the causes of arthritis by provocative techniques is accurate and clinically effective is to see if application of this information leads to a treatment that cures or significantly relieves an active case or prevents its recurrence. That is exactly what clinical ecologists have done for many years in case after case and study after study with thousands of patients. We do not claim to be able to cure the bone damage associated with arthritis if the allergy-initiated inflammation and structural changes are allowed to continue too long, but we can give considerable relief to many. Together, the arthritic and the ecologist often cure arthritis by continuous attention to the demonstrable causes in each case, while the condition remains in its reversible state. Careful avoidance of test-identified food offenders in conjunction with a rotary-diversified diet, desensitization treatment (immunotherapy), and changes in life-style, along with nutritional supplements, can prevent the occurrence of future “spontaneous” flare-ups due to exposure to previously unsuspected dietary and environmental factors.*15/295/5*

COMBATTING ASTHMA IN CHILDREN: ANTI-INFLAMMATORY DRUGS – STEROIDS: THE CORTISONE DRUGS – SIDE EFFECTS OF STEROID TREATMENT

The side effects which may arise with steroid treatment depend on the level of the dose and the duration for which it is taken. Major side effects take months to develop. Steroid treatment for a few days, or a few weeks, is safe and rarely causes problems. However, if steroids are used regularly several times a day, the chances of developing side effects increase. Many of the unacceptable side effects of cortisone can be avoided by giving the drug once a day in the morning, or on alternate days. Alternate-day steroid therapy has become a well-accepted method for treating asthma which does not respond to the conventional drugs. It has allowed many severe asthmatics to stay out of hospital and lead a relatively normal life. Prednisone or prednisolone is the cortisone drug of choice in alternate day therapy. However, sometimes children may develop the following fairly serious side effects after long-term use of oral steroids in higher doses.
1. Changes in shape of the body. Steroids often stimulate appetite, therefore weight gain is a common side effect. Steroids can also change the normal distribution of body fat, shifting it to the face and trunk, so that a round-faced, round-bodied look develops. This effect can be reversed when the drug is reduced or stopped.
2. Reduced growth rate. Prolonged use of steroids can interfere with the growth rate of the long bones of the body and this can affect the growth of a child. However, even after long periods of suppression by steroids, growth usually resumes when steroids are reduced or stopped.
3. Adrenal suppression. The adrenal glands are the body’s natural source of Cortisol. When cortisol-like drugs are given for extended periods of time, the adrenals may become ‘lazy’ and stop their own production of Cortisol. If the drug is discontinued abruptly after a long period of time, it takes time for the adrenals to begin to function again. During this interim period, the natural supply of corticosteroids is very low. This problem can be avoided by taking gradually decreasing doses. This gives the adrenal glands time to renew their production of Cortisol.
*61\260\8*

COMBATTING ASTHMA IN CHILDREN: ANTI-INFLAMMATORY DRUGS – STEROIDS: THE CORTISONE DRUGS – SIDE EFFECTS OF STEROID TREATMENTThe side effects which may arise with steroid treatment depend on the level of the dose and the duration for which it is taken. Major side effects take months to develop. Steroid treatment for a few days, or a few weeks, is safe and rarely causes problems. However, if steroids are used regularly several times a day, the chances of developing side effects increase. Many of the unacceptable side effects of cortisone can be avoided by giving the drug once a day in the morning, or on alternate days. Alternate-day steroid therapy has become a well-accepted method for treating asthma which does not respond to the conventional drugs. It has allowed many severe asthmatics to stay out of hospital and lead a relatively normal life. Prednisone or prednisolone is the cortisone drug of choice in alternate day therapy. However, sometimes children may develop the following fairly serious side effects after long-term use of oral steroids in higher doses.1. Changes in shape of the body. Steroids often stimulate appetite, therefore weight gain is a common side effect. Steroids can also change the normal distribution of body fat, shifting it to the face and trunk, so that a round-faced, round-bodied look develops. This effect can be reversed when the drug is reduced or stopped.2. Reduced growth rate. Prolonged use of steroids can interfere with the growth rate of the long bones of the body and this can affect the growth of a child. However, even after long periods of suppression by steroids, growth usually resumes when steroids are reduced or stopped.3. Adrenal suppression. The adrenal glands are the body’s natural source of Cortisol. When cortisol-like drugs are given for extended periods of time, the adrenals may become ‘lazy’ and stop their own production of Cortisol. If the drug is discontinued abruptly after a long period of time, it takes time for the adrenals to begin to function again. During this interim period, the natural supply of corticosteroids is very low. This problem can be avoided by taking gradually decreasing doses. This gives the adrenal glands time to renew their production of Cortisol.*61\260\8*

KEY POINTS: NEPHROPATHY

Nephropathy has traditionally occurred in 30-40% of people with type 1 diabetes.
Mortality from complications of nephropathy in type 1 diabetes was 80% in 1971, 40% in 1984, and 20% in 1996. With modern intensive management of glucose, blood pressure, albuminuria, and the synthesis and/or action of angiotensin II, the incidence of nephropathy and its cardiovascular complications will continue to decrease.
Intensive glucose management, with a HbAlc goal of < 7%, will decrease occurrence of microalbuminuria and subsequent diabetic nephropathy.
If microalbuminuria (> 30 mg/24 hr) is present, therapy with angiotensin-converting enzyme inhibitors leads to regression of microalbuminuria and delays the progression to clinical albuminuria (> 300 mg/24 hr) and/or end-stage renal failure. These effects are seen in normotensive as well as hypertensive type 1 diabetic patients vvith microalbuminuria.
ACE inhibitor therapy in type 1 diabetic patients with clinical albuminuria and an elevated serum creatinine leads to a 48-50% reduction in the risk of doubling of serum creatinine or progression to end-stage renal disease
*66\357\8*

KEY POINTS: NEPHROPATHYNephropathy has traditionally occurred in 30-40% of people with type 1 diabetes.Mortality from complications of nephropathy in type 1 diabetes was 80% in 1971, 40% in 1984, and 20% in 1996. With modern intensive management of glucose, blood pressure, albuminuria, and the synthesis and/or action of angiotensin II, the incidence of nephropathy and its cardiovascular complications will continue to decrease.  Intensive glucose management, with a HbAlc goal of < 7%, will decrease occurrence of microalbuminuria and subsequent diabetic nephropathy.If microalbuminuria (> 30 mg/24 hr) is present, therapy with angiotensin-converting enzyme inhibitors leads to regression of microalbuminuria and delays the progression to clinical albuminuria (> 300 mg/24 hr) and/or end-stage renal failure. These effects are seen in normotensive as well as hypertensive type 1 diabetic patients vvith microalbuminuria.ACE inhibitor therapy in type 1 diabetic patients with clinical albuminuria and an elevated serum creatinine leads to a 48-50% reduction in the risk of doubling of serum creatinine or progression to end-stage renal disease*66\357\8*